Thoas Fioretos
Research team manager
Genetic profiling of a chondroblastoma-like osteosarcoma/malignant phosphaturic mesenchymal tumor of bone reveals a homozygous deletion of CDKN2A, intragenic deletion of DMD, and a targetable FN1-FGFR1 gene fusion
Author
Summary, in English
Conventional osteosarcoma is the most common primary malignancy of bone. This group of neoplasms is subclassified according to specific histological features, but hitherto there has been no correlation between subtype, treatment, and prognosis. By in-depth genetic analyses of a chondroblastoma-like osteosarcoma, we detect a genetic profile that is distinct from those previously reported in benign and malignant bone tumors. The overall genomic copy number profile was less complex than that typically associated with conventional osteosarcoma, and there was no activating point mutation in any of H3F3A, H3F3B, IDH1, IDH2, BRAF, or GNAS. Instead, we found a homozygous CDKN2A deletion, a DMD microdeletion and an FN1-FGFR1 gene fusion. The latter alteration has been described in phosphaturic mesenchymal tumor. This tumor type shares some morphological features with chondroblastoma-like osteosarcoma and we cannot rule out that the present case actually represents an FN1-FGFR1 positive malignant phosphaturic mesenchymal tumor of bone without osteomalacia.
Department/s
- Genetic chaos in aggressive cancer
- BioCARE: Biomarkers in Cancer Medicine improving Health Care, Education and Innovation
- Division of Clinical Genetics
- Translational Genomic and Functional Studies of Leukemia
Publishing year
2019-05-08
Language
English
Pages
731-736
Publication/Series
Genes, Chromosomes and Cancer
Volume
58
Issue
10
Document type
Journal article
Publisher
John Wiley & Sons Inc.
Topic
- Cancer and Oncology
- Medical Genetics
Status
Published
Research group
- Genetic chaos in aggressive cancer
- Translational Genomic and Functional Studies of Leukemia
ISBN/ISSN/Other
- ISSN: 1045-2257